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Medical hypothesis discovery and innovation in ophthalmology

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Glycosylation End-product Formation in Retinal Membranes, an Energy Alteration Injury

  • Viroj Wiwanitkit

Medical hypothesis discovery and innovation in ophthalmology, Vol. 1 No. 1 (2012), 1 March 2012 , Page 18
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Abstract

Letter/ No abstract
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References

Kowluru RA. Diabetic retinopathy: mitochondrial dysfunction and retinal capillary cell death. Antioxid Redox Signal. 2005 Nov-Dec;7(11-12):1581-87. PMID: 16356121.

Santilli F, Cipollone F, Mezzetti A, Chiarelli F. The role of nitric oxide in the development of diabetic angiopathy. Horm Metab Res. 2004 May;36(5):319-35. PMID: 15156413.

Wiwanitkit V. Oxidation flux change in diabetic retinopathy: does it exist or not. Diabetol Croat. 2007;36(3):55-57.

Wiwanitkit V, House VWW, Bangkhae BT. Energy fluctuation in glycosylated end product formation: a new explanation on pathogenesis for complication in poorly controlled diabetic patient. Adv Lab Med Int 1. 2011;1(2):6-10.

Chen CL, Liang CM, Chen YH, Tai MC, Lu DW, Chen JT. Glucosamine modulates TNF-α-induced ICAM-1 expression and function through O-linked and N-linked glycosylation in human retinal pigment epithelial cells. Invest Ophthalmol Vis Sci. 2012 Apr 24;53(4):2281-91. PMID: 22427561.

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ISSN: 2322-3219

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